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Pregnancy & Hormones

The methylation-fertility

connection.

Nowhere are the stakes of methylation higher than in reproductive biology. Folate — specifically the bioactive form 5-MTHF — is required for DNA synthesis during rapid fetal cell division, and neural tube defects remain the most-cited consequence of inadequate folate in early pregnancy. For the ~30% of women who carry an MTHFR variant, the synthetic folic acid in most prenatal vitamins may not convert efficiently, making the pre-methylated form the safer bet.

Beyond pregnancy, methylation is the primary pathway by which the liver clears estrogen. Slow methylation → catechol estrogen accumulation → the symptom profile many women recognize as "estrogen dominance." COMT variants shape the downstream step and explain why some women metabolize hormones smoothly while others don't.

The articles below cover folate in pregnancy, MTHFR and miscarriage risk, and the estrogen-methylation connection.

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